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cox 1 2000 4850  (Cell Signaling Technology Inc)


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    Cell Signaling Technology Inc cox 1 2000 4850
    Cox 1 2000 4850, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 99/100, based on 11404 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/anti+cox+1+antibody/GAPDH+XP+Rabbit+mAb/bio_rxiv__64898__2026__02__26__708402-266-42-45
    Average 99 stars, based on 11404 article reviews
    cox 1 2000 4850 - by Bioz Stars, 2026-10
    99/100 stars

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    Related Articles

    Incubation:

    Article Title: Resolvin E1 and Resolvin E2 suppress cyclooxygenase-2 expression through ubiquitin-proteasome-mediated degradation in human macrophage-like U937 cells.
    Article Snippet: Inflammatory responses comprise a crucial defense mechanism against infection and injury.. Prostanoids, including prostaglandin E2 (PGE2), are well-known to play important roles in the generation of inflammatory responses.. However, their excessive or prolonged activation can cause tissue damage and drive the development of diseases.

    other:

    Article Title: Identification of a homozygous recessive variant in PTGS1 resulting in a congenital aspirin-like defect in platelet function
    Article Snippet: Protein concentration was quantified with a Bradford analysis and 25 μg of platelet lysates was loaded on a 10% Bis-Tris gel (BioRad, CA, USA).



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    QJSB alleviates inflammation in DSS-induced UC mice in vivo ( n = 6). (A) , (B) , (C) and (D) Levels of MPO, IL-6, IL-1β, and TNF-α in the colon; (E) , (F) and (G) serum levels of IL-1β, IL-6, and TNF-α; (H) levels of LPS in feces. (I) protein blots for iNOS and <t>COX-2;</t> (J) Relative expression levels of iNOS; (K) relative expression levels of COX-2; (L) correlation analysis between inflammatory mediators and colon-specific metabolites. (### p < 0.001, ## p < 0.01, # p vs. Control; * p < 0.05, ** p < 0.01, *** p < 0.001vs. Model).
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    Image Search Results


    QJSB alleviates inflammation in DSS-induced UC mice in vivo ( n = 6). (A) , (B) , (C) and (D) Levels of MPO, IL-6, IL-1β, and TNF-α in the colon; (E) , (F) and (G) serum levels of IL-1β, IL-6, and TNF-α; (H) levels of LPS in feces. (I) protein blots for iNOS and COX-2; (J) Relative expression levels of iNOS; (K) relative expression levels of COX-2; (L) correlation analysis between inflammatory mediators and colon-specific metabolites. (### p < 0.001, ## p < 0.01, # p vs. Control; * p < 0.05, ** p < 0.01, *** p < 0.001vs. Model).

    Journal: Frontiers in Microbiology

    Article Title: Qijiao Shengbai capsule ameliorates ulcerative colitis in mice by modulating gut microbiota and protecting the intestinal barrier, associated with suppression of the TLR4/MyD88/NF-κB pathway

    doi: 10.3389/fmicb.2026.1875650

    Figure Lengend Snippet: QJSB alleviates inflammation in DSS-induced UC mice in vivo ( n = 6). (A) , (B) , (C) and (D) Levels of MPO, IL-6, IL-1β, and TNF-α in the colon; (E) , (F) and (G) serum levels of IL-1β, IL-6, and TNF-α; (H) levels of LPS in feces. (I) protein blots for iNOS and COX-2; (J) Relative expression levels of iNOS; (K) relative expression levels of COX-2; (L) correlation analysis between inflammatory mediators and colon-specific metabolites. (### p < 0.001, ## p < 0.01, # p vs. Control; * p < 0.05, ** p < 0.01, *** p < 0.001vs. Model).

    Article Snippet: TLR4, MyD88, IκBα, p-IκBα, NF-κB p65, NF-κB p-p65, iNOS, and COX-2 antibodies were obtained from Proteintech (Wuhan, China).

    Techniques: In Vivo, Expressing, Analysis, Control